Posted by LAURA777 on February 8, 2003, at 16:44:16
In reply to Re: hey Bob » LAURA777, posted by not exactly on February 8, 2003, at 13:00:09
hey , well i think there may be truth to your theory . and the only reason i stated that cocaine was an ne agonist is because i read it on a nuerotransmitter synthesis site . they used it as an example that it was a good ne agonist but not a good anti-depressant ..
did you read that article about the opioids and genetics .. ??
i read about the crf factor in depression ,, cortisol levels and all .. hyper hypothalmic pituitary ..
can you explain this to me better , and what roll anti-depressants play . ?? it seems that they say that snri , helps with this ,?? i don't know , it was all pretty complicated ..
does norepinephrine have any thing at all to do with the amount of epinephrine in our systems ??
could there be a screw up some where where too much of this gets produced??? or could it possible be that a person can be super sensitive to it ????
are parkinsons drugs effective in raising the dopamine in the nucleus accumbens ?? do parkinsons drugs raise dopa throughout the brain or just in certain areas ???
sorry if you told me already , but did you try any of these ???
why does maoi's taken on long term have a likelyhood of causing high blood pressure ??? or was that tca's ??? sorry can't remember..i know i read somewhere that taking maoi's and tca's long term can cause problems .. ???
do you think that having a fast metabolisim and being very sensitive to stims have anything to do with the anhedonia ?? or how the nuerotransmiters work ??
what do you think ??
sorry for all the numerous questions .. i promise next time i will keep it down to only 4 questions !!! thanks for your time .. laura
poster:LAURA777
thread:138954
URL: http://www.dr-bob.org/babble/20030208/msgs/140194.html