Posted by LostBoyinNC1 on July 29, 2002, at 18:05:59
In reply to Re: Stop taking Zyprexa » Shawn. T., posted by CamW. on July 29, 2002, at 16:39:59
>
> As for 5-HT2 antagonism being responsible for a decreased incidence of EPS, this is probably wrong as well. Lack of EPS, and possibly a decreased incidence of raised prolactin levels, can be better explained by reversible D2 binding with the newer antipsychotics, as opposed to irreversible binding of conventional antipsychotics to D2 sites.If the newer atypical anti-psychotics dont cause EPS, how come all the ones I tried made my tongue numb, have muscle twitches and made my muscle everywhere become tight feeling? How come these drugs made me feel emotionally "flat?" How come all atypicals make my throat tight and I have mild trouble swallowing on them? How come last fall after a month on only 50 mg Seroquel my right side became weak and my psychiatrist found my right arm had become stiff and ratchety feeling and my right grip became weak? How come I began losing muscle tone on Seroquel Cam?
>
> The reason I have all of these articles is that I took a serious interest in this after a lecture by Phil Seeman I attended in 2000. At the time he was just formulating the theory. As a matter of fact, he had completely changed his lecture while talking with another presenter on the way from the airport. It was really cool to see the gears working in such an intelligent and passionate man.
Why do you believe all this drug company crap about atypical anti-psychotics Cam?
poster:LostBoyinNC1
thread:114125
URL: http://www.dr-bob.org/babble/20020725/msgs/114214.html